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Furthermore, P2X7-expressing MCs accumulate at the inflammatory sites in the colons of Crohn's disease patients.40 (2012) «Extracellular ATP mediates mast cell-dependent intestinal inflammation through P2X7 purinoceptors». Moreover, given that ATP promotes neutrophil migration47, it is possible that P2X7-dependent LT and chemokine production, as well as ATP generation via AK2 and ATP synthase from MCs, could amplify neutrophil infiltration of the colon. Our findings also suggest that MCs exacerbate inflammation by recruiting neutrophils to produce abundant TNF, but less IL-10 than is produced by other cells (for example, eosinophils, DCs and macrophages; Supplementary Fig. «S100 proteins in mouse and man: from evolution to function and pathology (including an update of the nomenclature. Bours MJ, Swennen EL, Di Virgilio F, Cronstein BN, Dagnelie PC (November 2006). Emberley ED, Murphy LC, Watson PH (August 2004). 1 2 (2015) «Immune Surveillance of the CNS following Infection and Injury».